A lack of predatory interaction between rumen ciliate protozoa and Shiga-toxin producing Escherichia coli
نویسندگان
چکیده
منابع مشابه
Shiga toxin-producing Escherichia coli
Shiga toxin-producing Escherichia coli (STEC) cause hemorrhagic colitis (HC) and hemolytic uremic syndrome (HUS) in humans. Outbreaks are linked to bovine food sources. STEC O157:H7 has been responsible for the most severe outbreaks worldwide. However, non-O157 serotypes have emerged as important enteric pathogens in several countries. The main virulence factor of STEC is the production of Shig...
متن کاملShiga Toxin – producing Escherichia coli
1. Rautio M, Lonnroth M, Saxen H, Nikku R, Väisanen ML, Finegold SM, et al. Characteristics of an unusual anaerobic pigmented gram-negative rod isolated from normal and infl amed appendices. Clin Infect Dis. 1997;25(Suppl 2):S107–10. 2. Rautio M, Saxen H, Siitonen A, Nikku R, Jousimies-Somer H. Bacteriology of histopathologically defi ned appendicitis in children. Pediatr Infect Dis J. 2000;19:...
متن کاملShiga Toxin–producing Escherichia coli, Idaho
1. Rautio M, Lonnroth M, Saxen H, Nikku R, Väisanen ML, Finegold SM, et al. Characteristics of an unusual anaerobic pigmented gram-negative rod isolated from normal and infl amed appendices. Clin Infect Dis. 1997;25(Suppl 2):S107–10. 2. Rautio M, Saxen H, Siitonen A, Nikku R, Jousimies-Somer H. Bacteriology of histopathologically defi ned appendicitis in children. Pediatr Infect Dis J. 2000;19:...
متن کاملShiga toxin-producing Escherichia coli: an overview.
The objective of this review is to highlight the importance of cattle in human disease due to Shiga toxin-producing Escherichia coli (STEC) and to discuss features of STEC that are important in human disease. Healthy dairy and beef cattle are a major reservoir of a diverse group of STEC that infects humans through contamination of food and water, as well as through direct contact. Infection of ...
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ژورنال
عنوان ژورنال: Letters in Applied Microbiology
سال: 2005
ISSN: 0266-8254,1365-2673
DOI: 10.1111/j.1472-765x.2004.01642.x